European Journal of Plant Pathology (2017) 148, 709-722

Xiumei Liu, Zhitian Zheng, Bin Li, Yiqiang Cai, Xuewei Mao and Mingguo Zhou (2017)
A myosin passenger protein gene (FaSmy1) is an essential regulator of cell development, pathogenicity, DON biosynthesis, and resistance to the fungicide phenamacril in Fusarium asiaticum
European Journal of Plant Pathology 148 (3), 709-722
Abstract: Kinesins, of which there are 650 members in 15 classes, have two main functions: cell separation and protein transport. In S. cerevisiae, the kinesin-like myosin passenger-protein Smy1(belonging to the TRAFAC class myosin-kinesin ATPase superfamily), which is transported by myosin-5,is part of a negative feedback mechanism that controls actin cable length and prevents overgrowth. The current study determined that defect deletion mutants of FaSmy1 had reduced development of hyphal growth, asexual spore production, pathogenicity, DON biosynthesis, and resistance to phenamacril. These results indicated that FaSmy1 is essential for the growth, development, reproduction and pathogenicity of F. asiaticum. FaSmy1 could be a useful target gene for development of new fungicides for control of Fusarium head blight.
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Link to article at publishers website
Database assignments for author(s): Ming-Guo Zhou

Research topic(s) for pests/diseases/weeds:
molecular biology - genes
pesticide resistance of pest

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Pest and/or beneficial records:

Beneficial	Pest/Disease/Weed	Crop/Product	Country	Quarant.
	Fusarium asiaticum